Lucíola Silva Barcelos: h-index, Total Citations, and Citation Map
Lucíola Silva Barcelos's h-index is 41 (76 i10-index, 4,836+ total citations across 5+ publications) according to Google Scholar as of May 2026. Lucíola Silva Barcelos is affiliated with Professor de Fisiologia, Universidade Federal de Minas Gerais.
Lucíola Silva Barcelos is a researcher affiliated with Professor de Fisiologia, Universidade Federal de Minas Gerais, specializing in Fisiologia vascular/Angiogênese, Inflamação, Células-tronco. Their work has been cited 4,836 times. This profile visualizes their global influence, highlighting strong citation networks in Brazil.
Lucíola Silva Barcelos's Citation Metrics
Bibliometric impact based on 5 indexed publications.
- H-Index
- 41
- i10-Index
- 76
- Total Citations
- 4,836
- Citing Countries
- 10
As of May 2026.
Lucíola Silva Barcelos has an h-index of 41 and 4,836 total citations across 5 publications, with research cited by institutions in 10 countries.
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Global Impact Map
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Top Cited Works
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Human CD133+ Progenitor Cells Promote the Healing of Diabetic Ischemic Ulcers by Paracrine Stimulation of Angiogenesis and Activation of Wnt Signaling
2009346
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Visa Evidence Package
Views and exports tuned for EB-1A, O-1A, and EB-2 NIW petitions. Sustained acclaim, geographic reach, and independent-citation filtering are the strongest evidence categories immigration adjudicators look for.
Significant Contributions
Auto-detected research lines — a seminal paper and the follow-up work building on it. Review and edit before using in a petition. Each Free PDF opens in a new tab — EB-1A organises this into the structure USCIS applies to Criterion 5 of 8 CFR § 204.5(h)(3)(v); EB-1B re-frames it under § 204.5(i)(3) (outstanding researcher); NIW presents it under prong 2 of Matter of Dhanasar.
The researcher established that human CD133+ progenitor cells heal diabetic ischemic ulcers via paracrine angiogenesis and Wnt signaling activation, a finding supported by over 300 citations.
The researcher identified elevated CCL2 and TNF-alpha levels in chagasic cardiomyopathy, establishing a key inflammatory mechanism in this disease through a seminal, highly cited study.
The researcher provided seminal evidence for the protective role of Angiotensin-(1–7) within the renin–angiotensin system in hepatic fibrosis, establishing a key mechanistic insight in liver disease pathophysiology.
Citation trend (last 10 years)Click to expand
Citation Trend (Last 10 Years)
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