Flávio Almeida Amaral: h-index, Total Citations, and Citation Map
Flávio Almeida Amaral's h-index is 42 (82 i10-index, 6,248+ total citations across 5+ publications) according to Google Scholar as of May 2026. Flávio Almeida Amaral is affiliated with Professor Adjunto, Universidade Federal de Minas Gerais.
Flávio Almeida Amaral is a researcher affiliated with Professor Adjunto, Universidade Federal de Minas Gerais, specializing in Imunologia. Their work has been cited 6,248 times. This profile visualizes their global influence, highlighting strong citation networks in China.
Flávio Almeida Amaral's Citation Metrics
Bibliometric impact based on 5 indexed publications.
- H-Index
- 42
- i10-Index
- 82
- Total Citations
- 6,248
- Citing Countries
- 12
As of May 2026.
Flávio Almeida Amaral has an h-index of 42 and 6,248 total citations across 5 publications, with research cited by institutions in 12 countries.
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Top Cited Works
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The CXCL8/IL-8 chemokine family and its receptors in inflammatory diseases
2014838
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Visa Evidence Package
Views and exports tuned for EB-1A, O-1A, and EB-2 NIW petitions. Sustained acclaim, geographic reach, and independent-citation filtering are the strongest evidence categories immigration adjudicators look for.
Significant Contributions
Auto-detected research lines — a seminal paper and the follow-up work building on it. Review and edit before using in a petition. Each Free PDF opens in a new tab — EB-1A organises this into the structure USCIS applies to Criterion 5 of 8 CFR § 204.5(h)(3)(v); EB-1B re-frames it under § 204.5(i)(3) (outstanding researcher); NIW presents it under prong 2 of Matter of Dhanasar.
The researcher provided a seminal synthesis of the CXCL8/IL-8 chemokine family and its receptors in inflammatory diseases, establishing a foundational reference for the field.
The researcher established that commensal microbiota is fundamental for the development of inflammatory pain, a finding supported by a seminal 2008 paper with 392 citations.
The researcher elucidated the mechanistic link between NLRP3 inflammasome activation and leukotriene B4-mediated neutrophil recruitment in gout, establishing a critical pathway for hypernociception.
Citation trend (last 10 years)Click to expand
Citation Trend (Last 10 Years)
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