Mehak Zahoor Khan: h-index, Total Citations, and Citation Map
Mehak Zahoor Khan's h-index is 11 (12 i10-index, 576+ total citations across 23+ publications) according to Google Scholar as of May 2026. Mehak Zahoor Khan is affiliated with Scientist III, Omniose | Research Fellow, Ragon Institute of MGH MIT and Harvard.
Mehak Zahoor Khan is a researcher affiliated with Scientist III, Omniose | Research Fellow, Ragon Institute of MGH MIT and Harvard, specializing in Immunology, Microbiology, Molecular Biology. Their work has been cited 576 times. This profile visualizes their global influence, highlighting strong citation networks in India.
Mehak Zahoor Khan's Citation Metrics
Bibliometric impact based on 23 indexed publications.
- H-Index
- 11
- i10-Index
- 12
- Total Citations
- 576
- Citing Countries
- 36
As of May 2026.
Mehak Zahoor Khan has an h-index of 11 and 576 total citations across 23 publications, with research cited by institutions in 36 countries.
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Top Cited Works
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Protein kinase G confers survival advantage to Mycobacterium tuberculosis during latency-like conditions
2017123
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Visa Evidence Package
Views and exports tuned for EB-1A, O-1A, and EB-2 NIW petitions. Sustained acclaim, geographic reach, and independent-citation filtering are the strongest evidence categories immigration adjudicators look for.
Significant Contributions
Auto-detected research lines — a seminal paper and the follow-up work building on it. Review and edit before using in a petition. Each Free PDF opens in a new tab — EB-1A organises this into the structure USCIS applies to Criterion 5 of 8 CFR § 204.5(h)(3)(v); EB-1B re-frames it under § 204.5(i)(3) (outstanding researcher); NIW presents it under prong 2 of Matter of Dhanasar.
233 citing papers could not be classified (no author data) — excluded from the percentages above.
The researcher elucidated the role of Protein kinase G in Mycobacterium tuberculosis survival during latency, establishing a foundational framework for understanding bacterial persistence mechanisms.
The researcher identified genome-wide non-CpG methylation patterns in host genomes during M. tuberculosis infection, establishing a novel epigenetic mechanism in host-pathogen interactions.
The researcher identified host sirtuin 2 as a novel immunotherapeutic target against tuberculosis, establishing a new paradigm for host-directed therapy in infectious disease research.
Citation trend (last 10 years)Click to expand
Citation Trend (Last 10 Years)
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