Fernando de Queiroz Cunha: h-index, Total Citations, and Citation Map
Fernando de Queiroz Cunha's h-index is 121 (645 i10-index, 56,190+ total citations across 5+ publications) according to Google Scholar as of May 2026. Fernando de Queiroz Cunha is affiliated with Professor Immunopharmacology, University of Sao Paulo.
Fernando de Queiroz Cunha is a researcher affiliated with Professor Immunopharmacology, University of Sao Paulo, specializing in Translacional Research Inflammatory Diseases. Their work has been cited 56,190 times. This profile visualizes their global influence, highlighting strong citation networks in United States.
Fernando de Queiroz Cunha's Citation Metrics
Bibliometric impact based on 5 indexed publications.
- H-Index
- 121
- i10-Index
- 645
- Total Citations
- 56,190
- Citing Countries
- 19
As of May 2026.
Fernando de Queiroz Cunha has an h-index of 121 and 56,190 total citations across 5 publications, with research cited by institutions in 19 countries.
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We've mapped 5,000 of 56,190 citations for Fernando de Queiroz Cunha
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Top Cited Works
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The pivotal role of tumour necrosis factor alpha in the development of inflammatory hyperalgesia
19921,051
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Visa Evidence Package
Views and exports tuned for EB-1A, O-1A, and EB-2 NIW petitions. Sustained acclaim, geographic reach, and independent-citation filtering are the strongest evidence categories immigration adjudicators look for.
Significant Contributions
Auto-detected research lines — a seminal paper and the follow-up work building on it. Review and edit before using in a petition. Each Free PDF opens in a new tab — EB-1A organises this into the structure USCIS applies to Criterion 5 of 8 CFR § 204.5(h)(3)(v); EB-1B re-frames it under § 204.5(i)(3) (outstanding researcher); NIW presents it under prong 2 of Matter of Dhanasar.
The researcher established the pivotal role of tumour necrosis factor alpha in inflammatory hyperalgesia through a seminal 1992 publication that has garnered over 1,000 citations.
The researcher elucidated the roles of resident macrophages and mast cells in zymosan- and acetic acid-induced writhing nociceptive responses in mice, establishing a foundational model for inflammatory pain mechanisms.
The researcher identified neutrophil extracellular traps as a key mediator of SARS-CoV-2 pathology, establishing a critical mechanistic link between immune response and disease severity.
Citation trend (last 10 years)Click to expand
Citation Trend (Last 10 Years)
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