Minxue Jia: h-index, Total Citations, and Citation Map
Minxue Jia's h-index is 10 (10 i10-index, 510+ total citations across 24+ publications) according to Google Scholar as of May 2026. Minxue Jia is affiliated with university of pittsburgh.
Minxue Jia is a researcher affiliated with university of pittsburgh, specializing in various fields. Their work has been cited 510 times. This profile visualizes their global influence, highlighting strong citation networks in United States.
Minxue Jia's Citation Metrics
Bibliometric impact based on 24 indexed publications.
- H-Index
- 10
- i10-Index
- 10
- Total Citations
- 510
- Citing Countries
- 38
As of May 2026.
Minxue Jia has an h-index of 10 and 510 total citations across 24 publications, with research cited by institutions in 38 countries.
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Global Impact Map
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Top Cited Works
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Myofibroblast transcriptome indicates SFRP2hi fibroblast progenitors in systemic sclerosis skin
2021278
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Top Citing Institutions
Visa Evidence Package
Views and exports tuned for EB-1A, O-1A, and EB-2 NIW petitions. Sustained acclaim, geographic reach, and independent-citation filtering are the strongest evidence categories immigration adjudicators look for.
Significant Contributions
Auto-detected research lines — a seminal paper and the follow-up work building on it. Review and edit before using in a petition. Each Free PDF opens in a new tab — EB-1A organises this into the structure USCIS applies to Criterion 5 of 8 CFR § 204.5(h)(3)(v); EB-1B re-frames it under § 204.5(i)(3) (outstanding researcher); NIW presents it under prong 2 of Matter of Dhanasar.
252 citing papers could not be classified (no author data) — excluded from the percentages above.
The researcher established a foundational link between SFRP2hi fibroblast progenitors and systemic sclerosis, subsequently expanding this framework to model chromatin dynamics in lung fibrosis and develop cross-modal computational tools for cell identification.
The researcher established a foundational link between natural killer cell dysfunction and idiopathic pulmonary fibrosis, subsequently expanding this framework to include monocyte transcriptomics and senescence mechanisms.
The researcher established a foundational framework for understanding transcriptional aging in the lung, subsequently extending this work to investigate lineage plasticity and develop computational tools for gene program dysregulation.
Citation trend (last 10 years)Click to expand
Citation Trend (Last 10 Years)
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