Jess Buxton: h-index, Total Citations, and Citation Map
Jess Buxton's h-index is 29 (41 i10-index, 12,952+ total citations across 5+ publications) according to Google Scholar as of May 2026. Jess Buxton is affiliated with Unknown affiliation.
Jess Buxton is a researcher affiliated with Unknown affiliation, specializing in Genetics, Telomeres, Epigenetics. Their work has been cited 12,952 times. This profile visualizes their global influence, highlighting strong citation networks in United States.
Jess Buxton's Citation Metrics
Bibliometric impact based on 5 indexed publications.
- H-Index
- 29
- i10-Index
- 41
- Total Citations
- 12,952
- Citing Countries
- 8
As of May 2026.
Jess Buxton has an h-index of 29 and 12,952 total citations across 5 publications, with research cited by institutions in 8 countries.
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We've mapped 5,000 of 12,952 citations for Jess Buxton
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Top Cited Works
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Molecular basis of myotonic dystrophy: expansion of a trinucleotide (CTG) repeat at the 3'end of a transcript encoding a protein kinase family member.
19923,510
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Visa Evidence Package
Views and exports tuned for EB-1A, O-1A, and EB-2 NIW petitions. Sustained acclaim, geographic reach, and independent-citation filtering are the strongest evidence categories immigration adjudicators look for.
Significant Contributions
Auto-detected research lines — a seminal paper and the follow-up work building on it. Review and edit before using in a petition. Each Free PDF opens in a new tab — EB-1A organises this into the structure USCIS applies to Criterion 5 of 8 CFR § 204.5(h)(3)(v); EB-1B re-frames it under § 204.5(i)(3) (outstanding researcher); NIW presents it under prong 2 of Matter of Dhanasar.
The researcher identified the molecular basis of myotonic dystrophy by linking CTG repeat expansions to a protein kinase transcript, a foundational discovery with over 3,500 citations.
The researcher identified a specific unstable DNA fragment associated with myotonic dystrophy, establishing a critical molecular marker for the disease.
The researcher identified and cloned the essential genomic region for myotonic dystrophy, mapping the putative defect and establishing a foundational genetic target for the disease.
Citation trend (last 10 years)Click to expand
Citation Trend (Last 10 Years)
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