Cahir O'Kane: h-index, Total Citations, and Citation Map
Cahir O'Kane's h-index is 54 (94 i10-index, 21,764+ total citations across 5+ publications) according to Google Scholar as of May 2026. Cahir O'Kane is affiliated with Department of Genetics, University of Cambridge.
Cahir O'Kane is a researcher affiliated with Department of Genetics, University of Cambridge, specializing in Drosophila Neurobiology and Neurodegeneration. Their work has been cited 21,764 times. This profile visualizes their global influence, highlighting strong citation networks in United States.
Cahir O'Kane's Citation Metrics
Bibliometric impact based on 5 indexed publications.
- H-Index
- 54
- i10-Index
- 94
- Total Citations
- 21,764
- Citing Countries
- 22
As of May 2026.
Cahir O'Kane has an h-index of 54 and 21,764 total citations across 5 publications, with research cited by institutions in 22 countries.
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Top Cited Works
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Inhibition of mTOR induces autophagy and reduces toxicity of polyglutamine expansions in fly and mouse models of Huntington disease
20042,955
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Visa Evidence Package
Views and exports tuned for EB-1A, O-1A, and EB-2 NIW petitions. Sustained acclaim, geographic reach, and independent-citation filtering are the strongest evidence categories immigration adjudicators look for.
Significant Contributions
Auto-detected research lines — a seminal paper and the follow-up work building on it. Review and edit before using in a petition. Each Free PDF opens in a new tab — EB-1A organises this into the structure USCIS applies to Criterion 5 of 8 CFR § 204.5(h)(3)(v); EB-1B re-frames it under § 204.5(i)(3) (outstanding researcher); NIW presents it under prong 2 of Matter of Dhanasar.
The researcher established that mTOR inhibition induces autophagy to reduce polyglutamine toxicity in Huntington disease models, a seminal finding published in Nature Genetics.
The researcher pioneered in situ detection methods for genomic regulatory elements in Drosophila, establishing a foundational approach for visualizing gene regulation within cellular contexts.
The researcher developed a targeted method to eliminate synaptic transmission in Drosophila using tetanus toxin, establishing a critical tool for linking specific neural circuits to behavioral defects.
Citation trend (last 10 years)Click to expand
Citation Trend (Last 10 Years)
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