Ania Wronski: h-index, Total Citations, and Citation Map
Ania Wronski's h-index is 12 (13 i10-index, 746+ total citations across 18+ publications) according to Google Scholar as of May 2026. Ania Wronski is affiliated with Unknown affiliation.
Ania Wronski is a researcher affiliated with Unknown affiliation, specializing in various fields. Their work has been cited 746 times. This profile visualizes their global influence, highlighting strong citation networks in United States.
Ania Wronski's Citation Metrics
Bibliometric impact based on 18 indexed publications.
- H-Index
- 12
- i10-Index
- 13
- Total Citations
- 746
- Citing Countries
- 12
As of May 2026.
Ania Wronski has an h-index of 12 and 746 total citations across 18 publications, with research cited by institutions in 12 countries.
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Top Cited Works
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The SIRT2 deacetylase stabilizes Slug to control malignancy of basal-like breast cancer
2016123
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Visa Evidence Package
Views and exports tuned for EB-1A, O-1A, and EB-2 NIW petitions. Sustained acclaim, geographic reach, and independent-citation filtering are the strongest evidence categories immigration adjudicators look for.
Significant Contributions
Auto-detected research lines — a seminal paper and the follow-up work building on it. Review and edit before using in a petition. Each Free PDF opens in a new tab — EB-1A organises this into the structure USCIS applies to Criterion 5 of 8 CFR § 204.5(h)(3)(v); EB-1B re-frames it under § 204.5(i)(3) (outstanding researcher); NIW presents it under prong 2 of Matter of Dhanasar.
The researcher identified SLUG-regulated cell-state transitions as critical mechanisms driving both tissue regeneration and tumor initiation, establishing a key link between developmental biology and oncogenesis.
The researcher established that de novo GTP biosynthesis is critical for the virulence of the fungal pathogen Cryptococcus neoformans, identifying a key metabolic target for antifungal intervention.
The researcher identified SIRT2 deacetylase as a stabilizer of Slug, establishing a novel mechanism controlling malignancy in basal-like breast cancer.
Citation trend (last 10 years)Click to expand
Citation Trend (Last 10 Years)
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