Hugo M. Horlings: h-index, Total Citations, and Citation Map
Hugo M. Horlings's h-index is 63 (132 i10-index, 31,518+ total citations across 2+ publications) according to Google Scholar as of May 2026. Hugo M. Horlings is affiliated with MD PhD, Netherlands Cancer Institute, Antoni van Leeuwenhoek Hospital, Amsterdam, The Netherlands.
Hugo M. Horlings is a researcher affiliated with MD PhD, Netherlands Cancer Institute, Antoni van Leeuwenhoek Hospital, Amsterdam, The Netherlands, specializing in Molecular pathology, therapy resistance, oncogenic pathways. Their work has been cited 31,518 times. This profile visualizes their global influence, highlighting strong citation networks in United States.
Hugo M. Horlings's Citation Metrics
Bibliometric impact based on 2 indexed publications.
- H-Index
- 63
- i10-Index
- 132
- Total Citations
- 31,518
- Citing Countries
- 10
As of May 2026.
Hugo M. Horlings has an h-index of 63 and 31,518 total citations across 2 publications, with research cited by institutions in 10 countries.
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We've mapped 5,000 of 31,518 citations for Hugo M. Horlings
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Top Cited Works
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Long non-coding RNA HOTAIR reprograms chromatin state to promote cancer metastasis
20106,226
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Visa Evidence Package
Views and exports tuned for EB-1A, O-1A, and EB-2 NIW petitions. Sustained acclaim, geographic reach, and independent-citation filtering are the strongest evidence categories immigration adjudicators look for.
Significant Contributions
Auto-detected research lines — a seminal paper and the follow-up work building on it. Review and edit before using in a petition. Each Free PDF opens in a new tab — EB-1A organises this into the structure USCIS applies to Criterion 5 of 8 CFR § 204.5(h)(3)(v); EB-1B re-frames it under § 204.5(i)(3) (outstanding researcher); NIW presents it under prong 2 of Matter of Dhanasar.
The researcher identified the PI3K pathway as a major determinant of trastuzumab resistance in breast cancer using a functional genetic approach.
The researcher established that the long non-coding RNA HOTAIR reprograms chromatin state to promote cancer metastasis, a seminal finding supported by over 6,000 citations.
Citation trend (last 10 years)Click to expand
Citation Trend (Last 10 Years)
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