Douglas Easton: h-index, Total Citations, and Citation Map
Douglas Easton's h-index is 222 (990 i10-index, 229,989+ total citations across 3+ publications) according to Google Scholar as of May 2026. Douglas Easton is affiliated with Professor of Genetic Epidemiology, University of Cambridge.
Douglas Easton is a researcher affiliated with Professor of Genetic Epidemiology, University of Cambridge, specializing in Genetic Epidemiology, Cancer Epidemiology, Statistical Genetics. Their work has been cited 229,989 times. This profile visualizes their global influence, highlighting strong citation networks in United States.
Douglas Easton's Citation Metrics
Bibliometric impact based on 3 indexed publications.
- H-Index
- 222
- i10-Index
- 990
- Total Citations
- 229,989
- Citing Countries
- 21
As of May 2026.
Douglas Easton has an h-index of 222 and 229,989 total citations across 3 publications, with research cited by institutions in 21 countries.
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We've mapped 5,000 of 229,989 citations for Douglas Easton
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Top Cited Works
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Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls
20079,923
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Visa Evidence Package
Views and exports tuned for EB-1A, O-1A, and EB-2 NIW petitions. Sustained acclaim, geographic reach, and independent-citation filtering are the strongest evidence categories immigration adjudicators look for.
Significant Contributions
Auto-detected research lines — a seminal paper and the follow-up work building on it. Review and edit before using in a petition. Each Free PDF opens in a new tab — EB-1A organises this into the structure USCIS applies to Criterion 5 of 8 CFR § 204.5(h)(3)(v); EB-1B re-frames it under § 204.5(i)(3) (outstanding researcher); NIW presents it under prong 2 of Matter of Dhanasar.
The researcher identified the BRCA2 gene as a breast cancer susceptibility factor, a foundational discovery that established critical genetic markers for hereditary cancer risk assessment.
The researcher conducted a large-scale genome-wide association study linking genetic variants to seven common diseases, establishing a foundational resource for complex trait genetics.
The researcher conducted a seminal combined analysis of 22 studies to establish average breast and ovarian cancer risks for BRCA1/2 mutations detected in unselected case series.
Citation trend (last 10 years)Click to expand
Citation Trend (Last 10 Years)
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