Mingchong Yang: h-index, Total Citations, and Citation Map
Mingchong Yang's h-index is 19 (21 i10-index, 994+ total citations across 29+ publications) according to Google Scholar as of June 2026. Mingchong Yang is affiliated with University of Connecticut Health Center.
Mingchong Yang is a researcher affiliated with University of Connecticut Health Center, specializing in Molecular Biology, Immunology, Stem Cell Biology. Their work has been cited 994 times. This profile visualizes their global influence, highlighting strong citation networks in China.
Mingchong Yang's Citation Metrics
Bibliometric impact based on 29 indexed publications.
- H-Index
- 19
- i10-Index
- 21
- Total Citations
- 994
- Citing Countries
- 55
As of June 2026.
Mingchong Yang has an h-index of 19 and 994 total citations across 29 publications, with research cited by institutions in 55 countries.
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Global Impact Map
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Scavenger receptor C mediates phagocytosis of white spot syndrome virus and restricts virus proliferation in shrimp
2016106
Top Citing Countries
Top Citing Institutions
Visa Evidence Package
Views and exports tuned for EB-1A, O-1A, and EB-2 NIW petitions. Sustained acclaim, geographic reach, and independent-citation filtering are the strongest evidence categories immigration adjudicators look for.
Significant Contributions
Auto-detected research lines — a seminal paper and the follow-up work building on it. Review and edit before using in a petition. Each Free PDF opens in a new tab — EB-1A organises this into the structure USCIS applies to Criterion 5 of 8 CFR § 204.5(h)(3)(v); EB-1B re-frames it under § 204.5(i)(3) (outstanding researcher); NIW presents it under prong 2 of Matter of Dhanasar.
65 citing papers could not be classified (no author data) — excluded from the percentages above.
The researcher established the role of scavenger receptors in shrimp antiviral immunity, identifying specific receptors that mediate phagocytosis and restrict White Spot Syndrome Virus proliferation.
The researcher established a framework linking complement proteins and vitamin D-binding protein in cerebrospinal fluid to multiple sclerosis activity and progression.
The researcher established a pharmacological strategy to activate tissue-specific mitophagy via USP30 inhibition, subsequently validating its therapeutic potential in cardiomyopathy models and human iPSC-derived cells.
Citation trend (last 10 years)Click to expand
Citation Trend (Last 10 Years)
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